Then, with recognition of the abstract schemas dictating how genes are expressed, biology climbed even closer to mathematics.

And then again, sometimes a single gene can influence how multiple traits are going to be expressed, and these genes are called pleiotropic.

Well, our genes are not our fate, and if we make these changes they're a predisposition but if we make bigger changes than we might have made otherwise, we can actually change how our genes are expressed.

Most longevity genes are master control genes, which extend lifespan by changing the activities of many subordinate genes and processes.

Resistance genes (R Genes) are genes in plant genomes that convey plant disease resistance against pathogens by producing R proteins.

These genes are the gene pool.

Are you more than your genes?

That's what your genes are doing.

So that suggests that somewhere in these genes, somewhere in the DNA, are genes for aging, genes that allow them to have different lifespans.

So that suggests that somewhere in these genes, somewhere in the DNA, are genes for aging, genes that allow them to have different lifespans.

Other genes that are not sex linked are also involved.

And on these chromosomes are roughly 25,000 genes.

That's what your genes are doing every day.

And be careful, your genes are in danger.

There are over a hundred genes that are associated with autism.

Most genes central in this model belong to the MADS box genes and are transcription factors that regulate the expression of the genes specific for each floral organ.

Dean Ornish says your genes are not your fate

And those genes are interesting, but they're very subtle.

Languages are genes talking, getting things that they want.

To some extent, they are programmed by your genes.

DNA repair genes are more active in these animals.

All told, you've nearly 20,000 genes, your genes are small parts of a long molecule, called DNA, or deoxyribonucleic acid.

In fact, most diseases are not caused by single genes.

Genes are the basic units of heredity in living organisms.

These are two genes that we found as good examples.

So, these genes are indeed going back to the populations.

Conversely, we know that new genes, designated ORFans ( orphan genes ), are commonly created by gene duplication, fusion, or other unknown mechanisms.

Portions of a DNA molecule that specify a single functional unit are called genes different genes have different sequences of bases.

These are things where genes are turned on in an individual cell type.

The idea that we're going to chose our children's genes, as we begin to understand what genes say about who we are.

And we found that over 500 genes were favorably changed in effect, turning on the good genes, the disease preventing genes, turning off the disease promoting genes.

That's how the same gene code that's why you have 30,000 genes, mice have 30,000 genes, husbands have 30,000 genes.

The proteins made by these genes are involved in repairing damaged DNA via the transcription coupled repair mechanism, particularly the DNA in active genes.

Genes are difficult to maintain, requiring subtle and energy intensive correcting processes.

Now, genes are short segments of DNA that code for amino acids.

He models genes.

Polygenic. Many genes.

Why do we live longer than dogs? The answer must lie in our genes, because genes are ultimately what make us different from other animals.

Genes, of course, are strings of DNA along the chromosome. It's currently thought that the human genome has about 25,000 genes which code for proteins.

And the genes it turns on includes antioxidant genes, genes I call carrot giver genes, whose protein products actually help other proteins to function well to fold correctly and function correctly.

And the genes it turns on includes antioxidant genes, genes I call carrot giver genes, whose protein products actually help other proteins to function well to fold correctly and function correctly.

While some researchers are looking for statistical correlations, others are studying how the individual genes interact.

In fact, we believe there are going to be something between 300 and 600 genes associated with autism, and genetic anomalies, much more than just genes.

Because it includes all mRNA transcripts in the cell, the transcriptome reflects the genes that are being actively expressed at any given time, with the exception of mRNA degradation phenomena such as transcriptional attenuation.

There are multiple genes that, when mutated, can cause the retinitis pigmentosa phenotype.